Thyroid Storm

Life-threatening exacerbation of thyrotoxicosis with multi-organ decompensation. Mortality ~10-30% even with treatment. Typically precipitated by infection, surgery, or trauma in uncontrolled hyperthyroidism. Characterised by high fever, tachycardia, agitation, and organ failure. Requires emergency multi-modal treatment.

Key Facts

Medical emergency: mortality ~10-30%; severe thyrotoxicosis with multi-organ dysfunction (cardiovascular, thermoregulatory, CNS, GI, hepatic) Precipitants: infection (commonest), surgery (especially thyroid surgery in unprepared patient), trauma, DKA, MI, drug withdrawal, radioiodine, iodinated contrast Clinical features: high fever (>38.5°C), severe tachycardia/AF/heart failure, agitation/delirium/psychosis/coma, nausea/vomiting/diarrhoea/jaundice, tremor, sweating Burch-Wartofsky Point Scale (BWPS): scoring system; >45 = thyroid storm likely; 25-44 = impending; <25 = unlikely Treatment (multi-modal, simultaneous): propylthiouracil 200mg QDS (blocks synthesis + T4→T3 conversion) or carbimazole 20-40mg QDS; Lugol iodine (1 hour after ATD — blocks thyroid hormone release); propranolol 40-80mg QDS (rate control, T4→T3 conversion); IV hydrocortisone 100mg QDS (reduces T4→T3, prevents adrenal crisis); supportive: cooling, IV fluids, treat precipitant

Overview

Key Facts

Thyroid storm is a clinical diagnosis based on the Burch-Wartofsky scale. Multi-modal treatment must be started immediately. The order of drug administration matters — give ATD before iodine.

Epidemiology

Rare: <2% of thyrotoxic patients. More common in Graves disease. Mortality ~10-30% even with treatment (higher in elderly, delayed treatment).

Aetiology

Almost always occurs on a background of uncontrolled/poorly treated hyperthyroidism with an acute precipitant: infection, surgery, trauma, DKA, MI, iodinated contrast, radioiodine, drug withdrawal.

Pathophysiology

Massive thyroid hormone excess → adrenergic hyperstimulation → multi-organ decompensation. Exact mechanism unclear — T4/T3 levels may not be dramatically higher than uncomplicated thyrotoxicosis; likely involves enhanced tissue sensitivity, cytokine surge, and precipitant-related stress response.

Clinical Presentation

Cardinal Features

  • Fever: high (>38.5°C, often >40°C); diaphoresis
  • Cardiovascular: severe tachycardia (>140 bpm), AF, high-output heart failure, hypotension/shock
  • CNS: agitation, delirium, psychosis, seizures, coma
  • GI: nausea, vomiting, diarrhoea, abdominal pain, jaundice (hepatic dysfunction — ominous sign)

Burch-Wartofsky Point Scale

  • Temperature, CNS effects, GI/hepatic, cardiovascular (HR, HF, AF) scored
  • 45 points = highly suggestive of thyroid storm

Red Flags

  • Multi-organ failure
  • Jaundice/hepatic dysfunction (poor prognostic sign)
  • Coma
  • Cardiovascular collapse

Differential Diagnosis

DiagnosisKey FeaturesInvestigation
SepsisFever, source of infection, raised lactateCultures, lactate
NMSAntipsychotic exposure, rigidity, raised CKDrug history, CK
Malignant hyperthermiaPost-anaesthesia, rigidity, raised CKGenetic testing
Phaeochromocytoma crisisParoxysmal HTN, headache, sweatingPlasma metanephrines
Heat strokeEnvironmental exposure, CNS dysfunctionHistory

Diagnosis / Investigation

Immediate

  • TFTs: suppressed TSH, markedly elevated T4/T3 (but may not be dramatically different from uncomplicated thyrotoxicosis)
  • FBC, U&Es, LFTs, coagulation, glucose, calcium
  • Blood cultures, urine cultures, CXR: identify precipitant
  • ECG: tachycardia, AF
  • ABG: metabolic/respiratory alkalosis or mixed

Burch-Wartofsky Scoring

  • Clinical scoring system to support diagnosis

Management

Emergency Multi-Modal Treatment (Simultaneous)

  1. Antithyroid drugs (block new hormone synthesis):

    • PTU 200mg QDS PO/NG preferred (also blocks peripheral T4→T3 conversion)
    • Alternative: carbimazole 20-40mg QDS PO/NG
  2. Iodine (blocks thyroid hormone release — give ≥1 HOUR after ATD):

    • Lugol iodine 5-10 drops TDS PO/NG
    • Or potassium iodide 60mg TDS
  3. Beta-blockers (rate control + T4→T3 block):

    • Propranolol 40-80mg QDS PO or 1-5mg IV slowly
    • Esmolol infusion if IV needed and cardiac monitoring available
  4. Corticosteroids (reduces T4→T3 conversion, treats relative adrenal insufficiency):

    • IV hydrocortisone 100mg QDS or IV dexamethasone 2mg QDS
  5. Supportive:

    • IV fluids, cooling measures (tepid sponging, fans; AVOID aspirin — displaces T4 from binding proteins)
    • Paracetamol for fever
    • Treat precipitant (antibiotics if infection)
    • Cardiac monitoring, ITU admission
    • Cholestyramine 4g QDS: may help by binding thyroid hormone in GI tract

Definitive Treatment

  • Once stabilised: plan definitive treatment (radioiodine or thyroidectomy after euthyroid state achieved)

Referral Criteria

  • ITU: all suspected thyroid storm
  • Endocrinology: urgent consultation
  • Surgery: if thyroidectomy needed as definitive treatment

Prognosis

Mortality ~10-30% even with treatment. Prompt recognition and multi-modal treatment improve survival. Poor prognostic factors: older age, coma, jaundice/hepatic failure, cardiovascular collapse, delayed treatment. With modern ICU care and aggressive treatment, outcomes are improving. Survivors require definitive treatment of underlying thyrotoxicosis.

Other Relevant Information

Thyroid Storm Treatment — Order of Administration

StepDrugMechanism
1PTU 200mg QDSBlocks synthesis + T4→T3
2 (≥1h after ATD)Lugol iodineBlocks hormone release
3Propranolol 40-80mg QDSRate control + T4→T3 block
4Hydrocortisone 100mg QDST4→T3 block + adrenal support
5SupportiveCooling, fluids, treat precipitant

Burch-Wartofsky Point Scale (Simplified)

ParameterPoints
Temperature ≥38.5°C15-30
CNS effects (agitation-coma)10-30
GI/hepatic (diarrhoea-jaundice)10-20
Heart rate ≥1005-25
Heart failure (mild-severe)5-20
AF present10
>45 = thyroid storm likely