Hypocalcaemia

Serum adjusted calcium <2.1 mmol/L. Commonest causes are vitamin D deficiency and hypoparathyroidism (post-surgical). Presents with neuromuscular excitability — tetany, perioral paraesthesia, seizures. Severe hypocalcaemia with prolonged QT is a medical emergency requiring IV calcium gluconate.

Key Facts

Commonest causes: vitamin D deficiency (most common overall), hypoparathyroidism (post-surgical — most common acute cause), chronic kidney disease Clinical features: neuromuscular excitability — perioral/digital paraesthesia, carpopedal spasm, tetany, Chvostek sign, Trousseau sign, seizures, laryngospasm ECG: prolonged QT interval (risk of Torsades de Pointes) Acute treatment: symptomatic or Ca²⁺ <1.9 mmol/L → IV calcium gluconate 10% 10mL over 10 minutes (with cardiac monitoring) → infusion; check and replace magnesium (Mg²⁺ required for PTH action) Chronic treatment: vitamin D deficiency → cholecalciferol (loading + maintenance); hypoparathyroidism → alfacalcidol/calcitriol + oral calcium Always check: PTH (primary differentiator), phosphate (high → hypoparathyroidism/CKD; low → vitamin D deficiency), magnesium, vitamin D, ALP, renal function

Overview

Key Facts

The approach to hypocalcaemia is guided by PTH and phosphate levels. Vitamin D deficiency is extremely common in the UK (~20% of adults). Post-surgical hypocalcaemia is the commonest acute presentation. Always check magnesium.

Epidemiology

Vitamin D deficiency: ~20% of UK adults have 25-OH-D <25 nmol/L. Post-thyroidectomy hypocalcaemia: transient ~10-30%, permanent ~1-2%.

Aetiology

Low PTH (hypoparathyroidism): post-surgical, autoimmune, DiGeorge, hypomagnesaemia. High PTH (secondary HPT): vitamin D deficiency (commonest), CKD, malabsorption (coeliac, IBD), pseudohypoparathyroidism. Other: pancreatitis (Ca²⁺ saponification), rhabdomyolysis, massive blood transfusion (citrate chelates Ca²⁺), hyperphosphataemia (tumour lysis), alkalosis (increased protein binding → lower ionised Ca²⁺).

Pathophysiology

Calcium homeostasis: PTH, vitamin D, and calcitonin regulate serum calcium through bone resorption, renal reabsorption, and intestinal absorption. Deficiency of PTH or vitamin D → reduced calcium from all three sources. Low ionised calcium → increased neuronal excitability → tetany, seizures. Cardiac effects: prolonged QT interval.

Clinical Presentation

Acute

  • Perioral paraesthesia: earliest symptom
  • Digital paraesthesia: fingers, toes
  • Muscle cramps, carpopedal spasm
  • Tetany: sustained involuntary muscle contraction
  • Laryngospasm: stridor — emergency
  • Seizures: generalised tonic-clonic
  • Chvostek sign: facial nerve tap → facial twitch
  • Trousseau sign: BP cuff → carpopedal spasm (more specific)

Chronic

  • Fatigue, depression, anxiety
  • Dry skin, brittle nails, coarse hair
  • Dental enamel hypoplasia
  • Cataracts
  • Basal ganglia calcification
  • Osteomalacia (vitamin D deficiency — bone pain, proximal myopathy)

ECG

  • Prolonged QT interval

Red Flags

  • Post-surgical perioral tingling → urgent calcium check
  • Laryngospasm/stridor → emergency
  • Seizure with hypocalcaemia → IV calcium
  • Prolonged QT → cardiac monitoring

Differential Diagnosis

DiagnosisKey FeaturesInvestigation
Vitamin D deficiencyHIGH PTH, LOW phosphate, raised ALP25-OH vitamin D
HypoparathyroidismLOW PTH, HIGH phosphatePTH
CKDHIGH PTH, HIGH phosphate, low eGFReGFR, PTH
PseudohypoparathyroidismHIGH PTH, Albright phenotypePTH, genetic testing
HypomagnesaemiaImpairs PTH functionSerum Mg²⁺
Acute pancreatitisAbdominal pain, raised amylaseAmylase, lipase

Diagnosis / Investigation

Essential

  • Adjusted calcium: <2.1 mmol/L
  • PTH: LOW = hypoparathyroidism; HIGH = secondary HPT (vitamin D deficiency, CKD)
  • Phosphate: high (hypoparathyroidism, CKD) vs low (vitamin D deficiency)
  • Magnesium: check and replace if low
  • 25-OH vitamin D: deficiency (<25 nmol/L), insufficiency (25-50 nmol/L)
  • ALP: raised in vitamin D deficiency/osteomalacia (normal in hypoparathyroidism)
  • U&Es: renal function (CKD)
  • Albumin: for adjusted calcium calculation

Other

  • ECG: prolonged QT
  • X-ray: Looser zones (pseudofractures) in osteomalacia
  • DEXA: if osteoporosis suspected

Management

Acute Severe Hypocalcaemia

  • IV calcium gluconate 10%: 10mL (2.25 mmol) IV over 10 minutes → repeat if needed → continuous infusion (40mL in 500mL 0.9% NaCl over 24 hours)
  • Cardiac monitoring: during IV calcium (risk of arrhythmia)
  • IV magnesium: magnesium sulphate 8 mmol IV over 20 minutes if Mg²⁺ low
  • Avoid IV calcium chloride peripherally (tissue necrosis if extravasation; use centrally if needed)
  • Transition to oral calcium + vitamin D as soon as possible

Vitamin D Deficiency

  • Loading: colecalciferol 50,000 IU weekly for 6 weeks (or equivalent); or fixed loading of 300,000 IU as split doses
  • Maintenance: colecalciferol 800-2000 IU daily (NICE/SACN)
  • Oral calcium supplementation if dietary intake inadequate
  • Recheck 25-OH-D at 3 months

Hypoparathyroidism

  • See separate topic: alfacalcidol/calcitriol + oral calcium

CKD

  • Active vitamin D analogues (alfacalcidol/calcitriol)
  • Phosphate binders
  • Manage CKD-MBD as per NICE NG203

Referral Criteria

  • Endocrinology: hypoparathyroidism, refractory hypocalcaemia
  • Nephrology: CKD-related
  • Gastroenterology: malabsorption

Prognosis

Vitamin D deficiency: excellent with supplementation; calcium normalises within weeks. Post-surgical hypoparathyroidism: transient resolves in days-weeks; permanent requires lifelong treatment. Chronic hypoparathyroidism: good quality of life achievable with monitoring. Severe acute hypocalcaemia: potentially fatal if untreated (laryngospasm, cardiac arrhythmia, seizures). Always responds to IV calcium.

Other Relevant Information

Approach to Hypocalcaemia

PTHPhosphateLikely Diagnosis
LOWHIGHHypoparathyroidism
HIGHLOWVitamin D deficiency
HIGHHIGHCKD, pseudohypoparathyroidism

IV Calcium Administration

PreparationRouteNotes
Calcium gluconate 10%Peripheral IVSafer; preferred
Calcium chloride 10%Central line ONLY3× more elemental calcium; tissue necrosis if extravasation