TextbookObstetrics & GynaecologyAntepartum Haemorrhage

Antepartum Haemorrhage

Antepartum haemorrhage (APH) is vaginal bleeding from 24 weeks gestation until delivery, complicating 3-5% of pregnancies and requiring urgent assessment for maternal and fetal wellbeing.

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Key Facts

APH is defined as vaginal bleeding from 24+0 weeks gestation until delivery Complicates 3-5% of pregnancies; most common causes are placenta praevia and placental abruption RCOG classification: minor (<50 mL), major (50-1000 mL), massive (>1000 mL or signs of shock) Never perform digital vaginal examination until placenta praevia has been excluded by ultrasound Anti-D immunoglobulin must be given to all RhD-negative women with APH (minimum 500 IU, Kleihauer test to guide dosing) Unexplained APH accounts for approximately 50% of cases Antenatal corticosteroids should be given if <34+6 weeks and delivery anticipated within 7 days APH is associated with increased risk of preterm delivery, FGR, and perinatal mortality

Overview

Key Facts

Antepartum haemorrhage is any bleeding from the genital tract from 24+0 weeks of pregnancy until delivery. It is a common obstetric emergency requiring systematic assessment to determine the cause, severity, and appropriate management.

Epidemiology

  • Complicates 3-5% of pregnancies
  • Accounts for significant proportion of obstetric emergencies
  • Placenta praevia: 0.5% of pregnancies at term
  • Placental abruption: 1% of pregnancies
  • Unexplained APH: approximately 50% of cases

Aetiology

  • Placenta praevia (low-lying placenta covering or near the cervical os)
  • Placental abruption (premature separation of normally sited placenta)
  • Vasa praevia (fetal vessels crossing the cervical os - rare but high fetal mortality)
  • Marginal bleeding (bleeding from placental edge)
  • Cervical causes: cervical ectropion, cervical cancer, cervical polyp
  • Infection: cervicitis, vaginitis
  • Unexplained (most common overall)

Pathophysiology

  • Placenta praevia: placental implantation in lower uterine segment; bleeding occurs as lower segment develops
  • Abruption: premature separation due to haemorrhage into the decidua basalis; may be concealed or revealed
  • Vasa praevia: rupture of unprotected fetal vessels, typically at membrane rupture

Clinical Presentation

Placenta Praevia Presentation

  • Painless bright red vaginal bleeding (classically recurrent)
  • Soft, non-tender uterus
  • Malpresentation common (breech, transverse lie)
  • Usually no fetal compromise unless massive bleeding

Placental Abruption Presentation

  • Constant abdominal pain with or without vaginal bleeding
  • Woody hard, tender uterus on palpation
  • Degree of shock may be disproportionate to visible blood loss (concealed haemorrhage)
  • Fetal distress or intrauterine fetal death

Vasa Praevia Presentation

  • Painless vaginal bleeding at membrane rupture
  • Rapid fetal compromise (sinusoidal CTG pattern)
  • Fetal mortality >50% if not diagnosed antenatally

Red Flags

  • Haemodynamic instability (tachycardia, hypotension)
  • Woody hard uterus (abruption)
  • Abnormal CTG
  • Bleeding at membrane rupture (vasa praevia)
  • Large volume blood loss (>500 mL)
  • Coagulopathy

Differential Diagnosis

DiagnosisKey FeaturesInvestigation
Placenta praeviaPainless bright red bleeding, soft uterusUltrasound (TVS)
Placental abruptionPainful, tense woody uterus, shockClinical diagnosis, USS may be normal
Vasa praeviaBleeding at membrane rupture, fetal distressAntenatal USS with colour Doppler
Cervical ectropionPostcoital bleeding, visualised on speculumSpeculum examination
Cervical cancerIrregular bleeding, visible lesionSpeculum, cervical biopsy
Bloody showSmall amount of blood-stained mucus, termClinical assessment
Uterine ruptureSevere pain, previous CS scar, shockClinical, laparotomy

Diagnosis / Investigation

Bedside

  • ABCDE assessment and haemodynamic monitoring
  • CTG (continuous fetal monitoring)
  • Speculum examination (DO NOT perform digital VE until praevia excluded)
  • Weigh pads/estimate blood loss
  • Group and save / crossmatch urgently

Bloods

  • FBC (haemoglobin, platelet count)
  • Coagulation screen (PT, APTT, fibrinogen)
  • Group and crossmatch (minimum 4 units in massive haemorrhage)
  • Kleihauer test (all RhD-negative women - quantifies fetomaternal haemorrhage)
  • U&Es, LFTs

Imaging

  • Transabdominal ultrasound: placental localisation, fetal assessment
  • Transvaginal ultrasound: gold standard for placental localisation (safe even with praevia)
  • Doppler assessment of fetal wellbeing

Special Tests

  • Apt test or haemoglobin electrophoresis if suspecting fetal blood (vasa praevia)
  • Fibronectin or cervical length if preterm labour suspected

Management

Non-pharmacological

  • ABCDE approach with simultaneous resuscitation
  • Two large-bore IV cannulae (14-16G)
  • Activate massive obstetric haemorrhage protocol if appropriate
  • Senior obstetric and anaesthetic involvement
  • Neonatal team alert

Pharmacological

  • Anti-D immunoglobulin: 500 IU IM minimum to all RhD-negative women; Kleihauer to guide additional doses
  • Antenatal corticosteroids: betamethasone 12mg IM × 2 (24h apart) if 24-34+6 weeks
  • Tocolysis: may be considered if preterm labour with mild APH (nifedipine 20mg PO); contraindicated with significant abruption
  • IV fluid resuscitation and blood product replacement as needed
  • Tranexamic acid 1g IV in massive haemorrhage

Surgical/Interventional

  • Placenta praevia: elective caesarean section at 36-37 weeks (RCOG GTG 27a)
  • Major abruption: emergency delivery (usually caesarean section) if fetal compromise
  • Vasa praevia: elective caesarean section at 34-36 weeks if diagnosed antenatally
  • Uterine compression sutures (B-Lynch), balloon tamponade, or uterine artery embolisation for ongoing haemorrhage

Referral Criteria

  • All APH requires hospital assessment
  • Consultant obstetrician review for any significant APH
  • Haematology input for coagulopathy
  • Blood bank alert for massive haemorrhage

Prognosis

  • Perinatal mortality varies by cause: placenta praevia (~1-2%), abruption (up to 30% in severe cases), vasa praevia (>50% if undiagnosed)
  • Maternal mortality rare in developed countries but remains significant worldwide
  • Recurrence risk: placenta praevia 4-8%, abruption 6-17%
  • APH associated with increased risk of preterm delivery, FGR, and neonatal morbidity
  • Women with unexplained APH have 2× risk of preterm delivery and 1.5× risk of low birth weight

Other Relevant Information

RCOG APH Severity Classification

CategoryBlood LossManagement
SpottingStaining onlyOutpatient if stable
Minor<50 mLAdmit, observe
Major50-1000 mLResuscitate, crossmatch
Massive>1000 mL or shockMajor haemorrhage protocol

Causes by Frequency

CauseApproximate Frequency
Unexplained50%
Placental abruption25-30%
Placenta praevia20%
Cervical causes5%
Vasa praevia<1%