TextbookNeurologyTension-Type Headache

Tension-Type Headache

Commonest primary headache disorder. Bilateral, pressing/tightening, mild-moderate intensity headache without significant nausea, photophobia, or phonophobia. Affects up to 80% of the population. Divided into episodic and chronic forms.

Key Facts

Commonest primary headache: lifetime prevalence up to 80%; accounts for ~90% of all headaches Bilateral, pressing/tightening ('band-like') quality; mild-moderate intensity; NOT aggravated by routine physical activity No nausea/vomiting (may have mild photophobia OR phonophobia but not both — distinguishes from migraine) Duration: 30 minutes to 7 days per episode Episodic (<15 days/month) and chronic (≥15 days/month for >3 months) forms Treatment: simple analgesia (paracetamol 1g, ibuprofen 400mg) for acute episodes; amitriptyline 10-75mg ON for chronic TTH prophylaxis (NICE CG150)

Overview

Key Facts

TTH is the most prevalent headache disorder globally. It is often underdiagnosed as patients may not seek medical attention. Chronic TTH can be significantly disabling and may overlap with medication overuse headache.

Epidemiology

Lifetime prevalence 30-80%. More common in women (F:M 1.5:1). Peak prevalence in 30s-40s. Chronic TTH affects ~3% of the population.

Aetiology

  • Multifactorial: stress, poor posture, anxiety, depression, sleep disruption, jaw clenching, eye strain
  • Chronic TTH: central sensitisation plays a key role
  • Genetic factors contribute (~40% concordance in twin studies)

Pathophysiology

Episodic TTH: peripheral mechanisms predominate — increased pericranial myofascial tenderness, nociceptive input from pericranial muscles. Chronic TTH: central sensitisation of second-order neurones in the trigeminal nucleus caudalis → enhanced pain perception; reduced descending inhibition. Peripheral and central mechanisms operate on a continuum from episodic to chronic TTH.

Clinical Presentation

Typical Features (ICHD-3)

  • Bilateral, pressing/tightening (non-pulsating) quality
  • Mild-moderate intensity (does NOT prevent activity — patients can continue work)
  • NOT aggravated by routine physical activity
  • Duration 30 minutes to 7 days
  • No nausea/vomiting (mild nausea allowed in chronic TTH)
  • Photophobia OR phonophobia (not both) or neither

Subtypes

  • Infrequent episodic: <1 day/month
  • Frequent episodic: 1-14 days/month for ≥3 months
  • Chronic: ≥15 days/month for >3 months

Associated Features

  • Pericranial muscle tenderness on palpation
  • Stress, anxiety, depression frequently coexist

Red Flags (Same SNOOP Criteria as Migraine)

  • Thunderclap onset, fever, focal neurological signs, papilloedema, new onset >50 years, progressive worsening

Differential Diagnosis

DiagnosisKey FeaturesInvestigation
MigraineUnilateral, pulsating, moderate-severe, nausea, photo+phonophobiaClinical (ICHD-3)
Medication overuse headacheDaily/near-daily, analgesic use ≥15 days/monthMedication diary
Cervicogenic headacheUnilateral, triggered by neck movement, occipital originClinical, MRI cervical spine
Idiopathic intracranial hypertensionPapilloedema, visual obscurations, pulsatile tinnitus, obese femaleLP (opening pressure), MRI/MRV
Giant cell arteritisAge >50, temporal tenderness, jaw claudication, raised ESRESR, CRP, temporal biopsy
Depression/anxietyPersistent headache with mood disturbancePsychiatric assessment

Diagnosis / Investigation

Clinical Diagnosis

  • TTH is a clinical diagnosis — no investigations needed for typical presentation
  • Headache diary: useful to classify frequency and identify medication overuse

When to Investigate

  • Red flag features: CT/MRI brain
  • New onset >50 years: ESR, CRP (exclude GCA)
  • Chronic daily headache: consider MRI to exclude secondary causes
  • Papilloedema: urgent CT then LP

Management

Acute Treatment

  • Paracetamol 1g or ibuprofen 400mg (or aspirin 600-900mg) — simple analgesia
  • Avoid codeine/opioid combinations (risk of medication overuse headache)
  • Limit acute treatment to <15 days/month (simple analgesics) or <10 days/month (triptans/opioids)

Prophylaxis (Chronic TTH)

  • Amitriptyline 10-75mg ON: first-line prophylactic; start at 10mg, titrate slowly; NICE CG150
  • Mirtazapine 15-30mg ON: alternative if amitriptyline not tolerated
  • Acupuncture: NICE CG150 recommends as option for chronic TTH prophylaxis

Non-Pharmacological

  • Stress management, relaxation techniques
  • CBT: effective for chronic TTH
  • Physiotherapy: cervical spine, posture correction
  • Regular exercise, adequate sleep hygiene
  • Reduce caffeine intake

Medication Overuse Headache

  • Withdraw overused medication (may need abrupt or gradual depending on agent)
  • Bridge with alternative if needed

Referral Criteria

  • Headache clinic: chronic TTH refractory to treatment, diagnostic uncertainty, medication overuse headache

Prognosis

Episodic TTH: generally benign, self-limiting. ~3% of episodic TTH progresses to chronic TTH per year. Chronic TTH: can be significantly disabling; ~40% remit to episodic over 2 years with treatment. Medication overuse is the most common factor perpetuating chronic TTH. Amitriptyline reduces headache frequency by ~50% in chronic TTH.

Other Relevant Information

Migraine vs Tension-Type Headache

FeatureMigraineTTH
LocationUnilateral (60%)Bilateral
QualityPulsatingPressing/tightening
IntensityModerate-severeMild-moderate
Physical activityAggravatedNot aggravated
Nausea/vomitingYesNo
Photophobia + phonophobiaBothNeither or one only
Duration4-72 hours30 min-7 days
Aura±No