TextbookOphthalmologyPosterior Uveitis

Posterior Uveitis

Posterior uveitis involves inflammation of the retina and/or choroid, commonly caused by toxoplasmosis, CMV, sarcoidosis, and tuberculosis, presenting with floaters, blurred vision, and potentially severe visual loss.

PLAB 1UKMLA1 questions

Key Facts

  • Posterior uveitis involves inflammation of the retina (retinitis), choroid (choroiditis), or both (chorioretinitis)
  • Toxoplasmosis is the most common infective cause of posterior uveitis worldwide; classic "headlight in fog" appearance on fundoscopy
  • CMV retinitis: occurs in immunocompromised (CD4 <50 in HIV); "pizza pie" fundus appearance; treat with ganciclovir/valganciclovir
  • Sarcoidosis: bilateral, granulomatous; "candle-wax drippings" (periphlebitis); elevated ACE, bilateral hilar lymphadenopathy
  • TB choroiditis: choroidal tubercles; consider in patients from endemic areas or with risk factors
  • Presents with floaters, blurred vision, and painless visual loss (unlike anterior uveitis)
  • Investigations: FBC, ESR, CRP, ACE, CXR, syphilis serology, QuantiFERON, toxoplasma serology, HIV test
  • Treatment depends on cause: antimicrobials for infection; immunosuppression for non-infectious causes

Overview

Key Facts

Posterior uveitis is less common than anterior uveitis but carries a higher risk of permanent visual loss. Identifying the underlying cause is essential for appropriate treatment.

Epidemiology

  • Accounts for 15-25% of all uveitis cases
  • Toxoplasmosis is the most common infective cause globally
  • CMV retinitis: rare since HAART but still occurs in immunocompromised
  • More likely to cause permanent visual loss than anterior uveitis

Aetiology

  • Infectious: toxoplasmosis, CMV, HSV/VZV, TB, syphilis, toxocara
  • Non-infectious: sarcoidosis, Behçet's disease, Vogt-Koyanagi-Harada (VKH), sympathetic ophthalmia, birdshot chorioretinopathy
  • Lymphoma (masquerade syndrome): can mimic chronic uveitis

Pathophysiology

  • Retinal/choroidal inflammation → tissue oedema → photoreceptor damage → visual loss
  • Vitreous cells and haze from inflammation
  • Macular oedema is the most common cause of visual loss in posterior uveitis
  • Retinal vasculitis may cause vessel occlusion and ischaemia

Clinical Presentation

Symptoms

  • Floaters (most common presenting symptom)
  • Blurred vision (may be painless)
  • Scotomata (visual field defects)
  • Usually no pain (unless associated anterior uveitis)
  • May be unilateral or bilateral

Signs

  • Vitreous cells and haze
  • Retinal or choroidal lesions on fundoscopy
  • Retinal vasculitis (sheathing of vessels)
  • Macular oedema
  • Specific patterns:
    • Toxoplasma: focal retinitis adjacent to old scar ("satellite lesion"); vitritis ("headlight in fog")
    • CMV: haemorrhagic retinal necrosis ("pizza pie")
    • Sarcoid: periphlebitis ("candle-wax drippings")

Red Flags

  • Rapidly progressive visual loss (CMV retinitis, acute retinal necrosis)
  • Immunocompromised patient (opportunistic infections)
  • Bilateral involvement (sarcoid, VKH, Behçet's)
  • Chronic vitritis not responding to treatment (consider lymphoma masquerade)

Differential Diagnosis

DiagnosisKey FeaturesInvestigation
ToxoplasmosisFocal retinitis near old scar, vitritisToxoplasma IgG/IgM, clinical
CMV retinitisImmunocompromised, haemorrhagic necrosisCD4 count, CMV PCR (aqueous)
SarcoidosisBilateral, periphlebitis, hilar lymphadenopathyACE, CXR, biopsy
TBChoroidal tubercles, risk factorsQuantiFERON, CXR
SyphilisGreat imitator; any pattern of uveitisSyphilis serology
Intraocular lymphomaChronic vitritis, poor response to steroidsVitreous biopsy, cytology

Diagnosis / Investigation

Bedside

  • Visual acuity (may be significantly reduced)
  • Fundoscopy (dilated): retinal/choroidal lesions, vitreous haze
  • Slit lamp: anterior segment involvement

Bloods

  • FBC, ESR, CRP
  • ACE, calcium (sarcoidosis)
  • Syphilis serology (VDRL/TPHA/FTA-Abs)
  • QuantiFERON-TB Gold / T-SPOT
  • Toxoplasma IgG/IgM
  • HIV test (if risk factors or CMV retinitis)

Imaging

  • CXR (sarcoidosis, TB)
  • OCT (macular oedema)
  • Fluorescein angiography (retinal vasculitis, ischaemia)
  • CT/MRI chest: sarcoidosis staging

Special Tests

  • Aqueous/vitreous tap + PCR: for viral causes (CMV, HSV, VZV, toxoplasma)
  • Vitreous biopsy: if lymphoma suspected
  • Indocyanine green angiography (ICG): choroidal lesions

Management

Non-pharmacological

  • MDT approach: ophthalmology, infectious diseases, respiratory (sarcoid/TB), rheumatology

Pharmacological

  • Toxoplasmosis: pyrimethamine 25-50mg + sulfadiazine 1g QDS + folinic acid (for 6-8 weeks); oral prednisolone added after 48h of antimicrobials
    • Alternative: co-trimoxazole + prednisolone
  • CMV retinitis: valganciclovir 900mg BD induction (21 days) then 900mg OD maintenance; intravitreal ganciclovir implant
    • Optimise ART in HIV patients
  • Sarcoidosis: topical/periocular/systemic steroids; steroid-sparing: methotrexate, azathioprine, mycophenolate; anti-TNFα (adalimumab)
  • TB: standard anti-TB treatment (RIPE) + corticosteroids (to prevent inflammatory damage)
  • Syphilis: IV benzylpenicillin 18-24 MU daily for 14 days (neurosyphilis regimen)
  • Non-infectious (general): systemic corticosteroids ± immunosuppression

Surgical/Interventional

  • Intravitreal steroid injections (triamcinolone, dexamethasone implant)
  • Vitrectomy: diagnostic (lymphoma) or therapeutic (severe vitritis, epiretinal membrane)
  • Laser: for retinal neovascularisation or ischaemia

Referral Criteria

  • All posterior uveitis: urgent ophthalmology referral
  • Immunocompromised with visual symptoms: same-day
  • Suspected intraocular lymphoma: urgent

Prognosis

  • Toxoplasmosis: good with treatment; recurrence in 50%; macular involvement causes permanent central visual loss
  • CMV retinitis: controlled with treatment but relapses common if immune suppression persists
  • Sarcoid uveitis: chronic relapsing; may require long-term immunosuppression; good visual prognosis if treated
  • Macular oedema: most common cause of visual loss; responds to treatment but may recur
  • Overall: posterior uveitis has worse visual prognosis than anterior uveitis

Other Relevant Information

Key Fundoscopic Appearances in Posterior Uveitis

CauseAppearance
ToxoplasmosisFocal white retinitis near old pigmented scar; "headlight in fog"
CMVHaemorrhagic retinal necrosis; "pizza pie" or "cottage cheese and ketchup"
SarcoidosisPeriphlebitis ("candle-wax drippings"); choroidal granulomas
TBChoroidal tubercles; serpiginous-like choroiditis
ARN (acute retinal necrosis)Peripheral retinal necrosis; HSV/VZV
BirdshotCream-coloured lesions scattered like birdshot; HLA-A29